Roles of noncoding RNAs in drug resistance in multiple myeloma
Author:
Affiliation:
1. Institute of Hematology, Union Hospital, Tongji Medical College Huazhong University of Science and Technology Wuhan China
2. Collaborative Innovation Center of Hematology Huazhong University of Science and Technology Wuhan China
Funder
National Natural Science Foundation of China
Publisher
Wiley
Subject
Cell Biology,Clinical Biochemistry,Physiology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/jcp.29726
Reference118 articles.
1. EZH2 inhibition in multiple myeloma downregulates myeloma associated oncogenes and upregulates microRNAs with potential tumor suppressor functions
2. miR-29b sensitizes multiple myeloma cells to bortezomib-induced apoptosis through the activation of a feedback loop with the transcription factor Sp1
3. Therapeutic Targeting of miR-29b/HDAC4 Epigenetic Loop in Multiple Myeloma
4. Drugging the lncRNA MALAT1 via LNA gapmeR ASO inhibits gene expression of proteasome subunits and triggers anti-multiple myeloma activity
5. Bortezomib action in multiple myeloma: microRNA-mediated synergy (and miR-27a/CDK5 driven sensitivity)?
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1. YY1-induced LncRNA-TUG1 elevates YOD1 to promote cell proliferation and inhibit bortezomib sensitivity in multiple myeloma;Leukemia & Lymphoma;2023-04-20
2. The Role of Non-coding RNAs in Drug Resistance of Cancers;Handbook of Cancer and Immunology;2023
3. Current perspectives on interethnic variability in multiple myeloma: Single cell technology, population pharmacogenetics and molecular signal transduction;Translational Oncology;2022-11
4. CircRNAs: novel therapeutic targets in multiple myeloma;Molecular Biology Reports;2022-06-21
5. Knockdown of lncRNA AL928768.3 inhibits multiple myeloma cell proliferation by inducing cell cycle arrest in G0/G1 phase;Annals of Translational Medicine;2022-02
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