Single and combined inhibition of tumor necrosis factor, interleukin-1, and RANKL pathways in tumor necrosis factor-induced arthritis: Effects on synovial inflammation, bone erosion, and cartilage destruction-Reference-Cited by-同舟云学术

Single and combined inhibition of tumor necrosis factor, interleukin-1, and RANKL pathways in tumor necrosis factor-induced arthritis: Effects on synovial inflammation, bone erosion, and cartilage destruction

Published:2004-01 Issue:1 Volume:50 Page:277-290
ISSN:0004-3591
Container-title:Arthritis & Rheumatism
language:en
Short-container-title:Arthritis & Rheumatism

Author:

Zwerina Jochen,Hayer Silvia,Tohidast-Akrad Makiyeh,Bergmeister Helga,Redlich Kurt,Feige Ulrich,Dunstan Colin,Kollias Giorgos,Steiner G�nter,Smolen Josef,Schett Georg

Publisher

Wiley

Subject

Pharmacology (medical),Immunology,Rheumatology,Immunology and Allergy

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1002/art.11487/fullpdf

Reference31 articles.

1. Repeated therapy with monoclonal antibody to tumour necrosis factor α (cA2) in patients with rheumatoid arthritis

2. Anti-tumor necrosis factor ameliorates joint disease in murine collagen-induced arthritis.

3. ROLE OF CYTOKINES IN RHEUMATOID ARTHRITIS

4. Transgenic mice expressing human tumour necrosis factor: a predictive genetic model of arthritis.

5. Adenovirus-based overexpression of tissue inhibitor of metalloproteinases 1 reduces tissue damage in the joints of tumor necrosis factor ? transgenic mice

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