Drug repositioning as an effective therapy for protease‐activated receptor 2 inhibition-Reference-Cited by-同舟云学术

Drug repositioning as an effective therapy for protease‐activated receptor 2 inhibition

Published:2018-10-29 Issue:2 Volume:120 Page:1522-1526
ISSN:0730-2312
Container-title:Journal of Cellular Biochemistry
language:en
Short-container-title:J Cell Biochem

Author:

Saqib Uzma¹^ORCID,Savai Rajkumar²^ORCID,Liu DongFang³⁴^ORCID,Banerjee Sreeparna⁵^ORCID,Baig Mirza S.⁶^ORCID

Affiliation:

1. Discipline of Chemistry Indian Institute of Technology Indore (IITI) Indore India

2. Department of Lung Development and Remodeling, Member of the German Center for Lung Research (DZL) Max Planck Institute for Heart and Lung Research Bad Nauheim Germany

3. Center for Inflammation & Epigenetics Houston Methodist Research Institute Houston Texas

4. Department of Microbiology and Immunology, Weill Cornell Medical College Cornell University New York

5. Department of Biological Sciences Orta Doğu Teknik Üniversitesi (ODTU/METU) Ankara Turkey

6. Discipline of Biosciences and Biomedical Engineering (BSBE) Indian Institute of Technology Indore (IITI) Indore India

Funder

Science and Engineering Research Board

Department of Biotechnology , Ministry of Science and Technology

Department of Science and Technology, Ministry of Science and Technology

Publisher

Wiley

Subject

Cell Biology,Molecular Biology,Biochemistry

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jcb.27334

Reference20 articles.

1. Molecular cloning, expression and potential functions of the human proteinase-activated receptor-2

2. Increased vascular permeability by a specific agonist of protease-activated receptor-2 in rat hindpaw

3. Characterization of the inflammatory response to proteinase-activated receptor-2 (PAR2)-activating peptides in the rat paw

4. Protease-Activated Receptor-2 Is Essential for Factor VIIa and Xa–Induced Signaling, Migration, and Invasion of Breast Cancer Cells

5. Trypsin Stimulates Integrin α5β1-dependent Adhesion to Fibronectin and Proliferation of Human Gastric Carcinoma Cells through Activation of Proteinase-activated Receptor-2