RAS/MAPK signaling functions in oxidative stress, DNA damage response and cancer progression-Reference-Cited by-同舟云学术

RAS/MAPK signaling functions in oxidative stress, DNA damage response and cancer progression

Published:2019-02-27 Issue:9 Volume:234 Page:14951-14965
ISSN:0021-9541
Container-title:Journal of Cellular Physiology
language:en
Short-container-title:J Cell Physiol

Author:

Rezatabar Setareh¹,Karimian Ansar¹²³,Rameshknia Vahid⁴⁵,Parsian Hadi¹,Majidinia Maryam⁶^ORCID,Kopi Tayebeh Azramezani¹,Bishayee Anupam⁷^ORCID,Sadeghinia Ali⁸,Yousefi Mehdi⁹^ORCID,Monirialamdari Mohsen⁵,Yousefi Bahman¹⁰¹¹^ORCID

Affiliation:

1. Cellular and Molecular Biology Research Center Health Research Institute, Babol University of Medical Sciences Babol Iran

2. Cancer & Immunology Research Center Kurdistan University of Medical Sciences Sanandaj Iran

3. Student Research Committee Babol University of Medical Sciences Babol Iran

4. Faculty of Medicine, Tabriz Branch Islamic Azad University Tabriz Iran

5. Immunology Research Center Tabriz University of Medical Sciences Tabriz Iran

6. Solid Tumor Research Center Urmia University of Medical Sciences Urmia Iran

7. Lake Erie College of Osteopathic Medicine Bradenton Florida

8. Department of Medicinal Chemistry, Faculty of Pharmacy Tabriz University of Medical Sciences Tabriz Iran

9. Drug Applied Research Center Tabriz University of Medical Sciences Tabriz Iran

10. Molecular Medicine Research Center Tabriz University of Medical Sciences Tabriz Iran

11. Department of Clinical Biochemistry and Laboratory Medicine, Faculty of Medicine Tabriz University of Medical Sciences Tabriz Iran

Publisher

Wiley

Subject

Cell Biology,Clinical Biochemistry,Physiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jcp.28334

Reference95 articles.

1. Ras Induces Chromosome Instability and Abrogation of the DNA Damage Response

2. Blocking Oncogenic Ras Signaling for Cancer Therapy

3. p53-dependent effects of RAS oncogene on chromosome stability and cell cycle checkpoints

4. Oxidative stress activates extracellular signal-regulated kinases through Src and Ras in cultured cardiac myocytes of neonatal rats.

5. Inhibition of caspase-9 through phosphorylation at Thr 125 by ERK MAPK

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