Implication of KCC2 in the sensitization to morphine by chronic ethanol treatment in mice
Author:
Affiliation:
1. Department of Toxicology; Hoshi University School of Pharmacy and Pharmaceutical Sciences, Shinagawa-Ku; Tokyo Japan
Publisher
Wiley
Subject
Cellular and Molecular Neuroscience
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1002/syn.21688/fullpdf
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3. Drugs abused by humans preferentially increase synaptic dopamine concentrations in the mesolimbic system of freely moving rats;Chiara;Proc Natl Acad Sci USA,1988
4. Neurosteroids, GABAA receptors, and ethanol dependence;Follesa;Psychopharmacology (Berl),2006
5. Suppression of ethanol-reinforced behavior by naltrexone is associated with attenuation of the ethanol-induced increase in dialysate dopamine levels in the nucleus accumbens;Gonzales;J Neurosci,1998
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1. Role of KCC2 in the Regulation of Brain-Derived Neurotrophic Factor on Ethanol Consumption in Rats;Molecular Neurobiology;2022-11-19
2. Protective Role of Low Ethanol Administration Following Ischemic Stroke via Recovery of KCC2 and p75NTR Expression;Molecular Neurobiology;2020-10-24
3. Treatment with zolpidem after ethanol administration potentiates the expression of ethanol-induced behavioral sensitization in mice;Brazilian Journal of Medical and Biological Research;2020
4. Splenectomy modifies hyperactive states of the dopaminergic system induced by morphine in C57BL/6J-bgJ/bgJ (beige-J) mice;European Journal of Pharmacology;2014-11
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