Hyperthermia engages the intrinsic apoptotic pathway by enhancing upstream caspase activation to overcome apoptotic resistance in MCF-7 breast adenocarcinoma cells-Reference-Cited by-同舟云学术

Hyperthermia engages the intrinsic apoptotic pathway by enhancing upstream caspase activation to overcome apoptotic resistance in MCF-7 breast adenocarcinoma cells

Published:2006 Issue:2 Volume:98 Page:356-369
ISSN:0730-2312
Container-title:Journal of Cellular Biochemistry
language:en
Short-container-title:J. Cell. Biochem.

Author:

Klostergaard J.,Leroux M.E.,Auzenne E.,Khodadadian M.,Spohn W.,Wu J.Y.,Donato N.J.

Publisher

Wiley

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference51 articles.

1. Analysis of Human Breast Adenocarcinoma MCF7 Resistance to Tumor Necrosis Factor-induced Cell Death

2. Selectivity of ceramide-mediated biology. Lack of activity of erythro-dihydroceramide.

3. Involvement of MACH, a Novel MORT1/FADD-Interacting Protease, in Fas/APO-1- and TNF Receptor–Induced Cell Death

4. Synergistic effect and possible mechanisms of tumor necrosis factor and cisplatin cytotoxicity under moderate hyperthermia against gastric cancer cells

5. FADD, a novel death domain-containing protein, interacts with the death domain of fas and initiates apoptosis

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