Frequent rearrangements of retinoic acid receptor alpha gene and myl gene, and rare mutations of RAS and FMS genes in acute promyelocytic leukemia
Author:
Publisher
Wiley
Subject
Hematology
Reference36 articles.
1. 15/17 TRANSLOCATION, A CONSISTENT CHROMOSOMAL CHANGE IN ACUTE PROMYELOCYTIC LEUKAEMIA
2. : “Catalog of Chromosome Aberrations in Cancer,” 3rd Ed. New York: Alan R. Liss; 1988.
3. The t(15;17) translocation of acute promyelocytic leukaemia fuses the retinoic acid receptor α gene to a novel transcribed locus
4. Molecular Analysis of Acute Promyelocytic Leukemia Breakpoint Cluster Region on Chromosome 17
5. Translocation breakpoint of acute promyelocytic leukemia lies within the retinoic acid receptor alpha locus.
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1. NRAS mutations are rare in acute myeloid leukaemias with t(8;21) or inv(16);European Journal of Haematology;2009-04-24
2. Secondary near-tetraploidy with double der(15)t(15;17) in acute promyelocytic leukemia in relapse;Cancer Genetics and Cytogenetics;2004-03
3. Fusion ofMLL andMSF in Adult De Novo Acute Myelomonocytic Leukemia (M4) with T(11;17)(Q23;Q25);International Journal of Hematology;2002-06
4. Anticoagulant effects of retinoic acids on leukemia cells;Blood;1996-01-15
5. The Biologic Function of PML and its Role in Acute Promyelocytic Leukemia;Leukemia & Lymphoma;1996-01
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