Increased aquaporin-1 and Na+-K+-2Cl- cotransporter 1 expression in choroid plexus leads to blood-cerebrospinal fluid barrier disruption and necrosis of hippocampal CA1 cells in acute rat models of hyponatremia-Reference-Cited by-同舟云学术

Increased aquaporin-1 and Na+-K+-2Cl- cotransporter 1 expression in choroid plexus leads to blood-cerebrospinal fluid barrier disruption and necrosis of hippocampal CA1 cells in acute rat models of hyponatremia

Published:2012-03-15 Issue:7 Volume:90 Page:1437-1444
ISSN:0360-4012
Container-title:Journal of Neuroscience Research
language:en
Short-container-title:J. Neurosci. Res.

Author:

Kim Jaehyun,Jung Yongwook

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1002/jnr.23017/fullpdf

Reference38 articles.

1. Volume changes induced by osmotic stress in freshly isolated rat hippocampal neurons;Aitken;Pflugers Arch,1998

2. Apoptosis. The role of the endonuclease;Arends;Am J Pathol,1990

3. Brain cell volume regulation in hyponatremia: role of sex, age, vasopressin, and hypoxia;Ayus;Am J Physiol Renal Physiol,2008

4. Effects of changes in serum osmolarity on bulk flow of fluid into cerebral ventricles and on brain water content;Di Mattio;Pflugers Arch,1975

5. Chronic hydrocephalus-induced hypoxia: increased expression of VEGFR-2+ and blood vessel density in hippocampus;Dombrowski;Neuroscience,2008

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