Isoform‐specific antibodies reveal distinct subcellular localizations of C 9orf72 in amyotrophic lateral sclerosis-Reference-Cited by-同舟云学术

Isoform‐specific antibodies reveal distinct subcellular localizations of C 9orf72 in amyotrophic lateral sclerosis

Published:2015-08-29 Issue:4 Volume:78 Page:568-583
ISSN:0364-5134
Container-title:Annals of Neurology
language:en
Short-container-title:Ann Neurol.

Author:

Xiao Shangxi¹,MacNair Laura¹²,McGoldrick Philip¹,McKeever Paul M.¹²,McLean Jesse R.¹,Zhang Ming¹,Keith Julia²³,Zinman Lorne²³,Rogaeva Ekaterina¹,Robertson Janice¹

Affiliation:

1. Tanz Center for Research in Neurodegenerative DiseasesUniversity of TorontoToronto Ontario Canada

2. Department of Laboratory Medicine and PathobiologyUniversity of TorontoToronto Ontario Canada

3. Sunnybrook Health Sciences CenterToronto Ontario Canada

Funder

Canadian Institutes of Health Research (JNM-90963)

Krembil Scientific Development Seed Fund

W. Garfield Weston Foundation

Temerty Family Foundation

James Hunter and Family ALS Initiative

Publisher

Wiley

Subject

Neurology (clinical),Neurology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/ana.24469

Reference48 articles.

1. Expanded GGGGCC Hexanucleotide Repeat in Noncoding Region of C9ORF72 Causes Chromosome 9p-Linked FTD and ALS

2. A Hexanucleotide Repeat Expansion in C9ORF72 Is the Cause of Chromosome 9p21-Linked ALS-FTD

3. Investigation of C9orf72 in 4 Neurodegenerative Disorders

4. Current insights into the C9orf72 repeat expansion diseases of the FTLD/ALS spectrum

5. The neuropathology associated with repeat expansions in the C9ORF72 gene

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