Exposure of tumor necrosis factor-α to luminal membrane of bovine brain capillary endothelial cells cocultured with astrocytes induces a delayed increase of permeability and cytoplasmic stress fiber formation of actin

Author:

Deli M. A.,Descamps L.,Dehouck M.-P.,Cecchelli R.,Joó F.,Ábrahám C. S.,Torpier G.

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience

Reference50 articles.

1. Signal transduction and the actin cytoskeleton: The roles of MARCKS and profilin;Aderem;TIBS,1992

2. Tumor necrosis factor in the pathogenesis of infectious diseases;Beutler;Crit Care Med [Suppl],1993

3. Growth and cultivation of dissociated neurones and glial cells from embryonic chick, rat and human brain in flask cultures;Booher;Neurobiology,1972

4. Tumor necrosis factor/cachectin increases permeability of endothelial cell monolayers by a mechanism involving regulatory G proteins;Brett;J Exp Med,1989

5. Tumor necrosis factor alters cytoskeletal organization and barrier function of endothelial cells;Camussi;Int Arch Allergy Appl Immunol,1991

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