Chronic opioid treatment of neuroblastoma X dorsal root ganglion neuron hybrid F11 cells results in elevated GM1 ganglioside and cyclic adenosine monophosphate levels and onset of naloxone-evoked decreases in membrane K+ currents-Reference-Cited by-同舟云学术

Chronic opioid treatment of neuroblastoma X dorsal root ganglion neuron hybrid F11 cells results in elevated GM1 ganglioside and cyclic adenosine monophosphate levels and onset of naloxone-evoked decreases in membrane K+ currents

Published:1995-11-01 Issue:4 Volume:42 Page:493-503
ISSN:0360-4012
Container-title:Journal of Neuroscience Research
language:en
Short-container-title:J. Neurosci. Res.

Author:

Wu G.,Fan S. F.,Lu Z.-H.,Ledeen R. W.,Crain S. M.

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience

Reference56 articles.

1. Possible Role of Gangliosides in Regulating an Adenylate Cyclase-Linked 5-Hydroxytryptamine (5-HT1) Receptor

2. An improved protein binding assay for cyclic AMP

3. Inhibitor of cyclic AMP-dependent protein kinase blocks opioid-induced prolongation of the action potential of mouse sensory ganglion neurons in dissociated cell cultures

4. Opioids can evoke direct receptor-mediated excitatory effects on sensory neurons

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