Ghrelin suppresses tunicamycin- or thapsigargin-triggered endoplasmic reticulum stress-mediated apoptosis in primary cultured rat cortical neuronal cells-Reference-Cited by-同舟云学术

Ghrelin suppresses tunicamycin- or thapsigargin-triggered endoplasmic reticulum stress-mediated apoptosis in primary cultured rat cortical neuronal cells

Published:2011 Issue:5 Volume:58 Page:409-420
ISSN:0918-8959
Container-title:Endocrine Journal
language:en
Short-container-title:Endocr J

Author:

Chung Hyunju¹,Chung Ho-Yeon¹,Bae Chong Woo¹,Kim Chong-Jin¹,Park Seungjoon²

Affiliation:

1. Department of Core Research Laboratory, Clinical Research Institute, The East-West Neo Medical Center, School of Medicine, Kyung Hee University, Gangdong-gu, Seoul, Korea

2. Department of Pharmacology and Medical Research Center for Bioreaction to ROS and Biomedical Science Institute, School of Medicine, Kyung Hee University, Seoul, 130-701, Korea

Publisher

Japan Endocrine Society

Subject

Endocrinology,Endocrinology, Diabetes and Metabolism

Link

http://www.jstage.jst.go.jp/article/endocrj/58/5/58_K10E-396/_pdf

Reference56 articles.

1. 1. Hashizume T, Horiuchi M, Nonaka S, Kasuya E, Kojima M, Hosoda H & Kangawa K (2005) Effects of ghrelin on growth hormone secretion in vivo in ruminants. Regul Pept 126: 61-65.

2. Ghrelin is a growth-hormone-releasing acylated peptide from stomach

3. A role for ghrelin in the central regulation of feeding

4. Ghrelin Inhibits Apoptosis in Hypothalamic Neuronal Cells during Oxygen-Glucose Deprivation

5. Phosphatidylinositol-3-kinase/Akt/glycogen synthase kinase-3β and ERK1/2 pathways mediate protective effects of acylated and unacylated ghrelin against oxygen–glucose deprivation-induced apoptosis in primary rat cortical neuronal cells

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