Lipase family member N is a novel target gene for CCAAT/enhancer-binding protein α in type 2 diabetic model mouse liver
Author:
Affiliation:
1. Faculty of Pharmaceutical Science, Fukuoka University, Fukuoka 814-0180, Japan
Publisher
Japan Endocrine Society
Subject
Endocrinology,Endocrinology, Diabetes and Metabolism
Link
https://www.jstage.jst.go.jp/article/endocrj/69/5/69_EJ21-0465/_pdf
Reference20 articles.
1. 1 Ramji DP, Foka P (2002) CCAAT/enhancer-binding proteins: structure, function and regulation. Biochem J 365: 561–575.
2. 2 Birkenmeier EH, Gwynn B, Howard S, Jerry J, Gordon JI, et al. (1989) Tissue-specific expression, developmental regulation, and genetic mapping of the gene encoding CCAAT/enhancer binding protein. Genes Dev 3: 1146–1156.
3. 3 Wang ND, Finegold MJ, Bradley A, Ou CN, Abdelsayed SV, et al. (1995) Impaired energy homeostasis in C/EBP alpha knockout mice. Science 269: 1108–1112.
4. 4 Inoue Y, Inoue J, Lambert G, Yim SH, Gonzalez FJ (2004) Disruption of hepatic C/EBPalpha results in impaired glucose tolerance and age-dependent hepatosteatosis. J Biol Chem 279: 44740–44748.
5. 5 Matsusue K, Gavrilova O, Lambert G, Brewer HB Jr, Ward JM, et al. (2004) Hepatic CCAAT/enhancer binding protein alpha mediates induction of lipogenesis and regulation of glucose homeostasis in leptin-deficient mice. Mol Endocrinol 18: 2751–2764.
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