A Nitric Oxide Synthesis Inhibitor Prevents the ACTH-Stimulated Production of Aldosterone in Rat Adrenal Gland.
Author:
Affiliation:
1. Second Department of Internal Medicine, Nihon University School of Medicine
Publisher
Japan Endocrine Society
Subject
Endocrinology,Endocrinology, Diabetes and Metabolism
Link
http://www.jstage.jst.go.jp/article/endocrj1993/43/2/43_2_157/_pdf
Cited by 15 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Glomerular and tubular effects of nitric oxide (NO) are regulated by angiotensin II (Ang II) in an age-dependent manner through activation of both angiotensin receptors (AT1Rs and AT2Rs) in conscious lambs;Pflügers Archiv - European Journal of Physiology;2017-08-31
2. Nitric Oxide at the Crossroad of Immunoneuroendocrine Interactions;Annals of the New York Academy of Sciences;2009-02
3. The rapid release of corticosterone from the adrenal induced by ACTH is mediated by nitric oxide acting by prostaglandin E2;Proceedings of the National Academy of Sciences;2005-04-18
4. Jew's Mellow Leaves (Corchorus olitorius) Suppress Elevation of Postprandial Blood Glucose Levels in Rats and Humans;International Journal for Vitamin and Nutrition Research;2005-01-01
5. Chronic administration of nitric oxide reduces angiotensin II receptor type 1 expression and aldosterone synthesis in zona glomerulosa cells;American Journal of Physiology-Endocrinology and Metabolism;2004-11
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