Attenuation of cell cycle progression by 2,3,7,8-tetrachlorodibenzo-p-dioxin eliciting ovulatory blockade in gonadotropin-primed immature rats
Author:
Affiliation:
1. Hormone Research Center and School of Biological Sciences & Technology, Chonnam National University, Kwangju, Republic of Korea
2. Animal Biotechnology Division, National Institute of Animal Science, Suwon, Republic of Korea
Publisher
Japan Endocrine Society
Subject
Endocrinology,Endocrinology, Diabetes and Metabolism
Link
http://www.jstage.jst.go.jp/article/endocrj/57/10/57_K10E-220/_pdf
Reference37 articles.
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2. 2. Rier SE (2008) Environmental immune disruption: a comorbidity factor for reproduction? Fertil Steril 89 (2 Suppl): e103-e108.
3. A review of mechanisms controlling ovulation with implications for the anovulatory effects of polychlorinated dibenzo-p-dioxins in rodents
4. 4. Sharara FI, Seifer DB, Flaws JA (1998) Environmental toxicants and female reproduction. Fertil Steril 70: 613-622.
5. Effects of Acute and Chronic Exposure to the Aryl Hydrocarbon Receptor Agonist 2,3,7,8-Tetrachlorodibenzo-p-Dioxin on the Transition to Reproductive Senescence in Female Sprague-Dawley Rats1
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