The role of the IL-23/IL-17 axis in the pathogenesis of Graves’ disease-Reference-Cited by-同舟云学术

The role of the IL-23/IL-17 axis in the pathogenesis of Graves’ disease

Published:2013 Issue:5 Volume:60 Page:591-597
ISSN:0918-8959
Container-title:Endocrine Journal
language:en
Short-container-title:Endocr J

Author:

Zheng Li¹,Ye Peng¹,Liu Chun¹

Affiliation:

1. Department of Endocrinology, The First Affiliated Hospital of Chongqing Medical University, Chongqing 400016, China

Publisher

Japan Endocrine Society

Subject

Endocrinology,Endocrinology, Diabetes and Metabolism

Link

https://www.jstage.jst.go.jp/article/endocrj/60/5/60_EJ12-0264/_pdf

Reference25 articles.

1. 1. Kolls JK, Linden (2004) A Interleukin-17 family members and inflammation. Immunity 21: 467-476.

2. 2. Chen Zijun, Liu Chun, Li Qiang, Tian Jie (2011) The change of Th17 lymphocytes and cytokines in autoimmune thyroid diseases. Immunological Journal 27: 49-52 (In Chinese).

3. 3. Li Qiang, Tian Jie, Chen Zijun, Li Xiafei, Liu Chun (2011) Oxidative stress up-zegnlale IL-17 in patients with Graves’ ophthalmopathy. J Third Mil Med Univ 33: 1068-1070 (In Chinese).

4. 4. Nanba T, Watanabe M, Inoue N, Iwatani Y, et al. (2009) Increases of the Th1/Th2 cell ratio in severe Hashimoto’s disease and in the proportion of Th17 cells in intractable Graves’ disease. Thyroid 19: 495-501.

5. 5. Ivanov II, McKenzie BS, Zhou L, Tadokoro CE, Lepelley A, et al. (2006) The orphan nuclear receptor RORγt directs the differentiation program of proinflammatory IL 17+Thelper cells. Cell 126: 1121-1133.

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