Functional polymorphisms affecting Th1 differentiation are associated with the severity of autoimmune thyroid diseases
Author:
Affiliation:
1. Department of Biomedical Informatics, Division of Health Sciences, Osaka University Graduate School of Medicine, Suita 565-0871, Japan
2. Laboratory for Clinical Investigation, Osaka University Hospital, Suita 565-0871, Japan
Publisher
Japan Endocrine Society
Subject
Endocrinology,Endocrinology, Diabetes and Metabolism
Link
https://www.jstage.jst.go.jp/article/endocrj/64/7/64_EJ16-0551/_pdf
Reference44 articles.
1. 1. Iwatani Y, Watanabe M (1999) Normal Mechanisms for self-tolerance. In: Volpe, R. (ed). Autoimmune endocrinopathies. Humana Press, Inc. Totawa: 1-30.
2. 2. Davies T (2000) Pathogenesis of Graves’ disease. In: Braverman LE., Utiger RD (ed). The thyroid - a Fundamental and Clinical Text. Lippincott Williams & Wilkins. Philadelphia: 518-531.
3. 3. Salgame P, Abrams JS, Clayberger C, Goldstein H, Convit J, et al. (1991) Differing lymphokine profiles of functional subsets of human CD4 and CD8 T cell clones. Science 254: 279-282.
4. 4. Mosmann TR, Sad S (1996) The expanding universe of T-cell subsets: Th1, Th2 and more. Immunol Today 17: 138-146.
5. 5. Abbas AK, Murphy KM, Sher A (1996) Functional diversity of helper T lymphocytes. Nature 383: 787-793.
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