Naive CD4 T Cells Highly Expressing the Inflammatory Chemokine Receptor CXCR3 Increase with Age and Radiation Exposure in Atomic Bomb Survivors
Author:
Affiliation:
1. Departments of Molecular Biosciences
2. Departments of Statistics
3. Departments of Clinical Studies
4. Departments of Epidemiology, Radiation Effects Research Foundation, Hiroshima
Publisher
Radiation Research Society
Subject
Radiology, Nuclear Medicine and imaging,Radiation,Biophysics
Link
https://meridian.allenpress.com/radiation-research/article-pdf/doi/10.1667/RADE-23-00065.1/3291850/10.1667_rade-23-00065.1.pdf
Reference24 articles.
1. G Desdin-Mico, G Soto-Heredero, JF Aranda, et al. "Science." 368, "T cells with dysfunctional mitochondria induce multimorbidity and premature senescence." 1371 (2020)
2. HM Cullings, EJ Grant, SD Egbert, et al. "Health Phys." 112, "DS02R1: Improvements to atomic bomb survivors' input data and implementation of Dosimetry System 2002 (DS02) and resulting changes in estimated doses." 56 (2017)
3. Y Kusunoki, M Yamaoka, F Kasagi, et al. "Radiat Res." 158, "T cells of atomic bomb survivors respond poorly to stimulation by Staphylococcus aureus toxins in vitro: does this stem from their peripheral lymphocyte populations having a diminished naive CD4 T-cell content?" 715 (2002)
4. Y Kusunoki, M Yamaoka, Y Kubo, et al. "Radiat Res." 174, "T-cell immunosenescence and inflammatory response in atomic bomb survivors." 870 (2010)
5. M Mittelbrunn and G Kroemer "Nat Immunol." 22, "Hallmarks of T cell aging." 687 (2021)
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