The role of p21/CIP1/WAF1 (p21) in the negative regulation of the growth hormone/growth hormone receptor and epidermal growth factor/epidermal growth factor receptor pathways, in growth hormone transduction defect
Author:
Publisher
Korean Society of Pediatric Endocrinology
Subject
Endocrinology, Diabetes and Metabolism,Pediatrics, Perinatology, and Child Health
Link
http://e-apem.org/upload/pdf/apem-2018-23-4-204.pdf
Reference20 articles.
1. Growth hormone/JAK-STAT axis signal-transduction defect.
2. Increased growth hormone receptor (GHR) degradation due to over-expression of cytokine inducible SH2 domain-containing protein (CIS) as a cause of GH transduction defect (GHTD)
3. Epidermal growth factor receptor (EGFR) involvement in successful growth hormone (GH) signaling in GH transduction defect
4. Cloning of p57KIP2, a cyclin-dependent kinase inhibitor with unique domain structure and tissue distribution.
5. Cell-cycle inhibition by independent CDK and PCNA binding domains in p21Cip1
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