Circulating plasmablasts contribute to antiphospholipid antibody production, associated with type I interferon upregulation
Author:
Publisher
Japanese Society on Thrombosis and Hemostasis
Subject
General Medicine
Link
https://www.jstage.jst.go.jp/article/jjsth/31/4/31_2020_JJTH_31_4_446-451/_pdf
Reference20 articles.
1. 1) Amengual O, Atsumi T: Antiphospholipid syndrome, “the best prophet of the future”. Mod Rheumatol 28: 409–416, 2018.
2. 2) Otomo K, Atsumi T, Amengual O, Fujieda Y, Kato M, Oku K, Horita T, Yasuda S, Koike T: Efficacy of the antiphospholipid score for the diagnosis of antiphospholipid syndrome and its predictive value for thrombotic events. Arthritis Rheum 64: 504–512, 2012.
3. 3) Bu C, Gao L, Xie W, Zhang J, He Y, Cai G, McCrae KR: beta2-glycoprotein i is a cofactor for tissue plasminogen activator-mediated plasminogen activation. Arthritis Rheum 60: 559–568, 2009.
4. 4) Zhang J, McCrae KR: Annexin A2 mediates endothelial cell activation by antiphospholipid/anti-beta2 glycoprotein I antibodies. Blood 105: 1964–1969, 2005.
5. 5) Arad A, Proulle V, Furie RA, Furie BC, Furie B: beta(2)-Glycoprotein-1 autoantibodies from patients with antiphospholipid syndrome are sufficient to potentiate arterial thrombus formation in a mouse model. Blood 117: 3453–3459, 2011.
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