Pathophysiology and treatment strategies for trauma induced DIC in acute phase of trauma
Author:
Affiliation:
1. 北海道大学病院救急科
Publisher
Japanese Society on Thrombosis and Hemostasis
Subject
General Medicine
Link
https://www.jstage.jst.go.jp/article/jjsth/33/5/33_2022_JJTH_33_5_535-543/_pdf
Reference63 articles.
1. 1) Rhee P, Joseph B, Pandit V, et al.: Increasing trauma deaths in the United States. Ann Surg 260: 13–21, 2014.
2. 2) Tisherman SA, Schmicker RH, Brasel KJ, et al.: Detailed description of all deaths in both the shock and traumatic brain injury hypertonic saline trials of the Resuscitation Outcomes Consortium. Ann Surg 261: 586–590, 2015.
3. 3) Hayakawa M, Maekawa K, Kushimoto S, et al.: Hyperfibrinolysis in severe isolated traumatic brain injury may occur without tissue hypoperfusion: A retrospective observational multicentre study. Crit Care 21: 222, 2017.
4. 4) Hayakawa M, Maekawa K, Kushimoto S, et al.: High D-dimer levels predict a poor outcome in patients with severe trauma, even with high fibrinogen levels on arrival: A multicenter retrospective study. Shock 45: 308–314, 2016.
5. 5) Hayakawa M, Gando S, Ono Y, et al.: Fibrinogen level deteriorates before other routine coagulation parameters and massive transfusion in the early phase of severe trauma: A retrospective observational study. Semin Thromb Hemost 41: 35–42, 2015.
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