Disulfram Treatment of NUP98-PHF23 AML Is Not EffectiveIn Vivo: Potential Role for Hematopoietic Stem Cells Niche
Author:
Affiliation:
1. Genetics Branch, Center for Cancer Research, National Cancer Institute/National Institute of Health, Bethesda, MD, USA
2. Department of Pediatrics and Institute of Health Science, College of Medicine, Gyeongsang National University, Jinju, Korea
Funder
Gyeongsang National University(GNU)
National Cancer Institute(NCI)
National Institutes of Health(NIH)
Publisher
The Korean Society of Pediatric Hematology-Oncology
Subject
General Materials Science
Link
http://pdf.medrang.co.kr/CPHO/2018/025/CPHO025-02-12.pdf
Reference17 articles.
1. NUP98 gene fusions and hematopoietic malignancies: common themes and new biologic insights
2. A novel NUP98-PHF23 fusion resulting from a cryptic translocation t(11;17)(p15;p13) in acute myeloid leukemia
3. Identification of the NUP98-PHF23 fusion gene in pediatric cytogenetically normal acute myeloid leukemia by whole-transcriptome sequencing
4. NUP98-PHF23 fusion is recurrent in acute myeloid leukemia and shares gene expression signature of leukemic stem cells
5. NUP98–PHF23 Is a Chromatin-Modifying Oncoprotein That Causes a Wide Array of Leukemias Sensitive to Inhibition of PHD Histone Reader Function
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