MYC and the unfolded protein response in cancer: synthetic lethal partners in crime?-Reference-Cited by-同舟云学术

MYC and the unfolded protein response in cancer: synthetic lethal partners in crime?

Published:2020-04-20 Issue:5 Volume:12 Page:
ISSN:1757-4676
Container-title:EMBO Molecular Medicine
language:en
Short-container-title:EMBO Mol Med

Author:

Zhang Tingting¹,Li Ningning²,Sun Chaoyang³^ORCID,Jin Yang⁴,Sheng Xia¹^ORCID

Affiliation:

1. Ministry of Education Key Lab of Environment and Health School of Public Health Tongji Medical College Huazhong University of Science and Technology Wuhan China

2. The Seventh Affiliated Hospital of Sun Yat‐sen University Shenzhen China

3. Department of Obstetrics and Gynecology Tongji Hospital Tongji Medical College Huazhong University of Science and Technology Wuhan China

4. Institute for Cancer Genetics and Informatics Oslo University Hospital Oslo Norway

Funder

Huazhong University of Science and Technology

National Natural Science Foundation of China

Helse Sør-Øst RHF

Publisher

EMBO

Subject

Molecular Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.15252/emmm.201911845

Reference127 articles.

1. Myc suppresses induction of the growth arrest genes gadd34, gadd45, and gadd153 by DNA-damaging agents

2. Characterization of a Novel PERK Kinase Inhibitor with Antitumor and Antiangiogenic Activity

3. Induction of CHOP Expression by Amino Acid Limitation Requires Both ATF4 Expression and ATF2 Phosphorylation

4. Discovery of GSK2656157: An Optimized PERK Inhibitor Selected for Preclinical Development

5. Mammalian Target of Rapamycin Complex 1 (mTORC1) Enhances Bortezomib-induced Death in Tuberous Sclerosis Complex (TSC)-null Cells by a c-MYC-dependent Induction of the Unfolded Protein Response

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