Treatment of hypertension by increasing impaired endothelial TRPV 4‐ KC a2.3 interaction-Reference-Cited by-同舟云学术

Treatment of hypertension by increasing impaired endothelial TRPV 4‐ KC a2.3 interaction

Published:2017-09-12 Issue:11 Volume:9 Page:1491-1503
ISSN:1757-4676
Container-title:EMBO Molecular Medicine
language:en
Short-container-title:EMBO Mol Med

Author:

He Dongxu¹²,Pan Qiongxi¹²,Chen Zhen¹²,Sun Chunyuan¹²,Zhang Peng¹²,Mao Aiqin¹²,Zhu Yaodan¹²,Li Hongjuan¹²,Lu Chunxiao¹²,Xie Mingxu¹²,Zhou Yin¹²,Shen Daoming¹²,Tang Chunlei¹²,Yang Zhenyu³,Jin Jian¹²,Yao Xiaoqiang⁴,Nilius Bernd⁵,Ma Xin¹²^ORCID

Affiliation:

1. School of Medicine Jiangnan University Wuxi China

2. National Engineering Laboratory for Cereal Fermentation Technology Jiangnan University Wuxi China

3. Heart Centre Wuxi People's Hospital Wuxi China

4. School of Biomedical Sciences The Chinese University of Hong Kong Shatin Hong Kong

5. Department Cell Mol Medicine Laboratory Ion Channel Research Campus Gasthuisberg KU Leuven Leuven Belgium

Funder

National Natural Science Foundation of China

Publisher

EMBO

Subject

Molecular Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.15252/emmm.201707725

Reference54 articles.

1. Isoform-Selective Physical Coupling of TRPC3 Channels to IP 3 Receptors in Smooth Muscle Cells Regulates Arterial Contractility

2. An Elevation in Physical Coupling of Type 1 Inositol 1,4,5-Trisphosphate (IP 3 ) Receptors to Transient Receptor Potential 3 (TRPC3) Channels Constricts Mesenteric Arteries in Genetic Hypertension

3. Low intravascular pressure activates endothelial cell TRPV4 channels, local Ca2+ events, and IKCa channels, reducing arteriolar tone

4. High-Salt Diet Enhances Vascular Reactivity in Pregnant Rats With Normal and Reduced Uterine Perfusion Pressure

5. Genetic Deficit of SK3 and IK1 Channels Disrupts the Endothelium-Derived Hyperpolarizing Factor Vasodilator Pathway and Causes Hypertension

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