Inflammatory monocytes promote progression of Duchenne muscular dystrophy and can be therapeutically targeted via CCR 2-Reference-Cited by-同舟云学术

Inflammatory monocytes promote progression of Duchenne muscular dystrophy and can be therapeutically targeted via CCR 2

Published:2014-10-13 Issue:11 Volume:6 Page:1476-1492
ISSN:1757-4676
Container-title:EMBO Molecular Medicine
language:en
Short-container-title:EMBO Mol Med

Author:

Mojumdar Kamalika¹,Liang Feng¹,Giordano Christian¹,Lemaire Christian¹,Danialou Gawiyou¹,Okazaki Tatsuma¹,Bourdon Johanne¹,Rafei Moutih²,Galipeau Jacques³⁴,Divangahi Maziar¹,Petrof Basil J¹

Affiliation:

1. Meakins‐Christie Laboratories and Respiratory Division McGill University Health Centre and Research Institute Montreal QC Canada

2. Department of Pharmacology Faculty of Medicine University of Montreal Montreal QC Canada

3. Department of Hematology and Oncology Winship Cancer Institute Emory University Atlanta GA USA

4. Department of Pediatrics Emory University Atlanta GA USA

Publisher

EMBO

Subject

Molecular Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.15252/emmm.201403967

Reference66 articles.

1. Interplay of IKK/NF-κB signaling in macrophages and myofibers promotes muscle degeneration in Duchenne muscular dystrophy

2. Infiltrating monocytes trigger EAE progression, but do not contribute to the resident microglia pool

3. The mdx-amplification-resistant mutation system assay, a simple and rapid polymerase chain reaction-based detection of the mdx allele

4. Inflammatory monocytes recruited after skeletal muscle injury switch into antiinflammatory macrophages to support myogenesis

5. Monitoring of Blood Vessels and Tissues by a Population of Monocytes with Patrolling Behavior

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