KLF 4 is a key determinant in the development and progression of cerebral cavernous malformations-Reference-Cited by-同舟云学术

KLF 4 is a key determinant in the development and progression of cerebral cavernous malformations

Published:2015-11-26 Issue:1 Volume:8 Page:6-24
ISSN:1757-4676
Container-title:EMBO Molecular Medicine
language:en
Short-container-title:EMBO Mol Med

Author:

Cuttano Roberto¹,Rudini Noemi¹,Bravi Luca¹,Corada Monica¹,Giampietro Costanza¹²,Papa Eleanna¹³,Morini Marco Francesco¹⁴,Maddaluno Luigi¹⁵,Baeyens Nicolas⁶,Adams Ralf H⁷,Jain Mukesh K⁸⁹¹⁰¹¹,Owens Gary K¹²,Schwartz Martin⁶,Lampugnani Maria Grazia¹¹³,Dejana Elisabetta¹¹⁴¹⁵

Affiliation:

1. IFOM the FIRC Institute of Molecular Oncology Milan Italy

2. Department of Biosciences University of Milan Milan Italy

3. on leave of absence at Department of Neurology Laboratory for Molecular Neuro‐Oncology University Hospital Zurich Zurich Switzerland

4. on leave of absence at Department of Biomedicine University of Basel Basel Switzerland

5. on leave of absence at Institute of Molecular Health Sciences ETH Zurich Zurich Switzerland

6. Yale Cardiovascular Research Center New Haven CT USA

7. Department of Tissue Morphogenesis Faculty of Medicine Max Planck Institute for Molecular Biomedicine University of Münster Münster Germany

8. Case Cardiovascular Research Institute Cleveland OH USA

9. Harrington Heart & Vascular Institute Cleveland OH USA

10. Department of Medicine University Hospitals Case Medical Center Cleveland OH USA

11. Case Western Reserve University School of Medicine University Hospitals Case Medical Center Cleveland OH USA

12. Robert M. Berne Cardiovascular Research Center University of Virginia School of Medicine Charlottesville Virginia USA

13. Mario Negri Institute of Pharmacological Research Milan Italy

14. Department of Immunology Genetics and Pathology Uppsala University Uppsala Sweden

15. Department of Oncology and Oncohematology University of Milan Milan Italy

Publisher

EMBO

Subject

Molecular Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.15252/emmm.201505433

Reference87 articles.

1. Deficiency for endoglin in tumor vasculature weakens the endothelial barrier to metastatic dissemination

2. Krit 1 interactions with microtubules and membranes are regulated by Rap1 and integrin cytoplasmic domain associated protein-1

3. Mutations within the Programmed Cell Death 10 Gene Cause Cerebral Cavernous Malformations

4. Rho Kinase Inhibition Rescues the Endothelial Cell Cerebral Cavernous Malformation Phenotype

5. Developmental timing of CCM2 loss influences cerebral cavernous malformations in mice

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