Pancreatic cancer microenvironment, to target or not to target?
Author:
Affiliation:
1. Department of Internal Medicine Mayo Clinic Rochester MN USA
2. Schulze Center for Novel Therapeutics Mayo Clinic Rochester MN USA
Publisher
EMBO
Subject
Molecular Medicine
Link
https://onlinelibrary.wiley.com/doi/pdf/10.15252/emmm.201505948
Reference13 articles.
1. Exclusion of T Cells From Pancreatic Carcinomas in Mice Is Regulated by Ly6Clow F4/80+ Extratumoral Macrophages
2. Compression of Pancreatic Tumor Blood Vessels by Hyaluronan Is Caused by Solid Stress and Not Interstitial Fluid Pressure
3. Targeting CXCL12 from FAP-expressing carcinoma-associated fibroblasts synergizes with anti-PD-L1 immunotherapy in pancreatic cancer
4. Hyaluronan impairs vascular function and drug delivery in a mouse model of pancreatic cancer
5. Macrophage-secreted cytokines drive pancreatic acinar-to-ductal metaplasia through NF-κB and MMPs
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