Oligonucleotide‐induced alternative splicing of serotonin 2C receptor reduces food intake-Reference-Cited by-同舟云学术

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Oligonucleotide‐induced alternative splicing of serotonin 2C receptor reduces food intake

Published:2016-07-12 Issue:8 Volume:8 Page:878-894
ISSN:1757-4676
Container-title:EMBO Molecular Medicine
language:en
Short-container-title:EMBO Mol Med

Author:

Zhang Zhaiyi¹,Shen Manli¹,Gresch Paul J²,Ghamari‐Langroudi Masoud³,Rabchevsky Alexander G⁴,Emeson Ronald B³,Stamm Stefan¹^ORCID

Affiliation:

1. Department of Molecular and Cellular Biochemistry University of Kentucky Lexington KY USA

2. Department of Pharmacology Vanderbilt University Nashville TN USA

3. Department of Molecular Physiology & Biophysics Vanderbilt University Nashville TN USA

4. Spinal Cord & Brain Injury Research Center University of Kentucky Lexington KY USA

Funder

National Institutes of Health

Foundation for Prader-Willi Research

National Institute of Child Health and Human Development

National Institute of General Medical Sciences

Publisher

EMBO

Subject

Molecular Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.15252/emmm.201506030

Reference50 articles.

1. Design principles of interconnections between chromatin and pre-mRNA splicing

2. Identification of rat serotonin 5-HT2C receptors as glycoproteins containing N-linked oligosaccharides

3. Characteristics of compounds that cross the blood-brain barrier

4. RNA Targeting Therapeutics: Molecular Mechanisms of Antisense Oligonucleotides as a Therapeutic Platform

5. Serotonin 2C receptors in pro-opiomelanocortin neurons regulate energy and glucose homeostasis

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