Smooth muscle FGF / TGF β cross talk regulates atherosclerosis progression-Reference-Cited by-同舟云学术

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Smooth muscle FGF / TGF β cross talk regulates atherosclerosis progression

Published:2016-05-13 Issue:7 Volume:8 Page:712-728
ISSN:1757-4676
Container-title:EMBO Molecular Medicine
language:en
Short-container-title:EMBO Mol Med

Author:

Chen Pei‐Yu¹,Qin Lingfeng²,Li Guangxin²³,Tellides George²,Simons Michael¹⁴^ORCID

Affiliation:

1. Department of Internal Medicine Yale Cardiovascular Research Center Yale University School of Medicine New Haven CT USA

2. Department of Surgery Yale University School of Medicine New Haven CT USA

3. Department of Vascular Surgery The First Hospital of China Medical University Shenyang China

4. Department of Cell Biology Yale University School of Medicine New Haven CT USA

Funder

National Institutes of Health

Publisher

EMBO

Subject

Molecular Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.15252/emmm.201506181

Reference48 articles.

1. Human smooth muscle myosin heavy chain isoforms as molecular markers for vascular development and atherosclerosis.

2. Phenotypic Modulation of Smooth Muscle Cells during Progression of Human Atherosclerosis as Determined by Altered Expression of Myosin Heavy Chain Isoforms

3. Smooth Muscle Cells in Atherosclerosis Originate From the Local Vessel Wall and Not Circulating Progenitor Cells in ApoE Knockout Mice

4. Distinct patterns of expression of fibroblast growth factors and their receptors in human atheroma and nonatherosclerotic arteries. Association of acidic FGF with plaque microvessels and macrophages.

5. Compensatory signalling induced in the yolk sac vasculature by deletion of TGFβ receptors in mice

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