Inhibition of O‐GlcNAcylation protects from Shiga toxin‐mediated cell injury and lethality in host-Reference-Cited by-同舟云学术

Inhibition of O‐GlcNAcylation protects from Shiga toxin‐mediated cell injury and lethality in host

Published:2021-11-29 Issue:1 Volume:14 Page:
ISSN:1757-4676
Container-title:EMBO Molecular Medicine
language:en
Short-container-title:EMBO Mol Med

Author:

Lee Kyung‐Soo¹²^ORCID,Lee Jieun¹,Lee Pureum¹²,Jeon Bong Chan²³,Song Min Yeong¹²,Kwak Sojung¹,Lee Jungwoon¹²,Kim Jun‐Seob⁴^ORCID,Kim Doo‐Jin⁵,Kim Ji Hyung⁵,Tesh Vernon L⁶,Lee Moo‐Seung¹²^ORCID,Park Sung‐Kyun⁵^ORCID

Affiliation:

1. Environmental Diseases Research Center Korea Research Institute of Bioscience & Biotechnology (KRIBB) Daejeon Korea

2. Department of Biomolecular Science KRIBB School of Bioscience Korea University of Science and Technology (UST) Daejeon Korea

3. Immunotherapy Convergence Research Center Korea Research Institute of Bioscience & Biotechnology (KRIBB) Daejeon Korea

4. Department of Nano‐Bioengineering Incheon National University Incheon Korea

5. Infectious Disease Research Center Korea Research Institute of Bioscience & Biotechnology (KRIBB) Daejeon Korea

6. Department of Microbial Pathogenesis and Immunology College of Medicine Texas A&M University Bryan TX USA

Funder

National Research Foundation of Korea

Korea Research Institute of Bioscience and Biotechnology

Publisher

EMBO

Subject

Molecular Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.15252/emmm.202114678

Reference78 articles.

1. Modification of RelA by O-linked N-acetylglucosamine links glucose metabolism to NF- B acetylation and transcription

2. Renal Subcapsular Transplantation of PSC-Derived Kidney Organoids Induces Neo-vasculogenesis and Significant Glomerular and Tubular Maturation In Vivo

3. O-GlcNAc Cycling: A Link Between Metabolism and Chronic Disease

4. Characterization of the Catalytic Domain of Clostridium novyi Alpha-Toxin

5. Glucosamine protects neonatal cardiomyocytes from ischemia-reperfusion injury via increased protein O-GlcNAc and increased mitochondrial Bcl-2

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