Ca M Kinase II mediates maladaptive post‐infarct remodeling and pro‐inflammatory chemoattractant signaling but not acute myocardial ischemia/reperfusion injury-Reference-Cited by-同舟云学术

Ca M Kinase II mediates maladaptive post‐infarct remodeling and pro‐inflammatory chemoattractant signaling but not acute myocardial ischemia/reperfusion injury

Published:2014-09-05 Issue:10 Volume:6 Page:1231-1245
ISSN:1757-4676
Container-title:EMBO Molecular Medicine
language:en
Short-container-title:EMBO Mol Med

Author:

Weinreuter Martin¹²,Kreusser Michael M¹²,Beckendorf Jan¹²,Schreiter Friederike C¹²,Leuschner Florian²³,Lehmann Lorenz H¹²,Hofmann Kai P¹²,Rostosky Julia S¹²,Diemert Nathalie¹²,Xu Chang¹²,Volz Hans Christian²³,Jungmann Andreas²³,Nickel Alexander⁴,Sticht Carsten⁵,Gretz Norbert⁵,Maack Christoph⁴,Schneider Michael D⁶,Gröne Hermann‐Josef⁷,Müller Oliver J²³,Katus Hugo A²³,Backs Johannes¹²

Affiliation:

1. Research Unit Cardiac Epigenetics Department of Cardiology University of Heidelberg Heidelberg Germany

2. DZHK (German Centre for Cardiovascular Research) Partner Site Heidelberg/Mannheim Heidelberg Germany

3. Department of Cardiology University of Heidelberg Heidelberg Germany

4. Department of Cardiology Saarland University Homburg Germany

5. Medical Research Center University of Heidelberg Medical Faculty Mannheim Mannheim Germany

6. British Heart Foundation Centre of Research Excellence Faculty of Medicine National Heart and Lung Institute Imperial College London London UK

7. Department of Cellular and Molecular Pathology German Cancer Research Center Heidelberg Germany

Publisher

EMBO

Subject

Molecular Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.15252/emmm.201403848

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4. CaM kinase II selectively signals to histone deacetylase 4 during cardiomyocyte hypertrophy

5. Histone Deacetylase 5 Acquires Calcium/Calmodulin-Dependent Kinase II Responsiveness by Oligomerization with Histone Deacetylase 4

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