DOT1L‐HES6 fusion drives androgen independent growth in prostate cancer
Author:
Affiliation:
1. Institute of Biosciences and Medical Technology Tampere Finland
2. Department of Signal Processing Tampere University of Technology Tampere Finland
3. Department of Pathology University of Texas M.D. Anderson Cancer Center Houston TX USA
Publisher
EMBO
Subject
Molecular Medicine
Link
https://onlinelibrary.wiley.com/doi/pdf/10.15252/emmm.201404210
Reference8 articles.
1. Glucocorticoid Receptor Confers Resistance to Antiandrogens by Bypassing Androgen Receptor Blockade
2. Molecular Characterization of Neuroendocrine Prostate Cancer and Identification of New Drug Targets
3. ChimeraScan: a tool for identifying chimeric transcription in sequencing data
4. Acheate-scute like 1 (Ascl1) is required for normal delta-like (Dll) gene expression and notch signaling during retinal development
5. HES6 drives a critical AR transcriptional programme to induce castration‐resistant prostate cancer through activation of an E 2 F 1‐mediated cell cycle network
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