A novel phosphocholine‐mimetic inhibits a pro‐inflammatory conformational change in C‐reactive protein-Reference-Cited by-同舟云学术

A novel phosphocholine‐mimetic inhibits a pro‐inflammatory conformational change in C‐reactive protein

Published:2022-12-05 Issue:1 Volume:15 Page:
ISSN:1757-4676
Container-title:EMBO Molecular Medicine
language:en
Short-container-title:EMBO Mol Med

Author:

Zeller Johannes¹²^ORCID,Cheung Tung Shing Karen S³⁴^ORCID,Nero Tracy L³⁴⁵^ORCID,McFadyen James D²⁴^ORCID,Krippner Guy²^ORCID,Bogner Balázs¹,Kreuzaler Sheena¹,Kiefer Jurij¹^ORCID,Horner Verena K¹,Braig David¹^ORCID,Danish Habiba²⁶,Baratchi Sara⁶^ORCID,Fricke Mark¹^ORCID,Wang Xiaowei²⁴^ORCID,Kather Michel G⁷^ORCID,Kammerer Bernd⁷^ORCID,Woollard Kevin J⁸^ORCID,Sharma Prerna²,Morton Craig J³⁴^ORCID,Pietersz Geoffrey²⁴^ORCID,Parker Michael W³⁴⁵^ORCID,Peter Karlheinz²⁴^ORCID,Eisenhardt Steffen U¹^ORCID

Affiliation:

1. Department of Plastic and Hand Surgery, University of Freiburg Medical Centre Medical Faculty of the University of Freiburg Freiburg Germany

2. Baker Heart and Diabetes Institute Melbourne Vic. Australia

3. Department of Biochemistry and Pharmacology, Bio21 Molecular Science and Biotechnology Institute The University of Melbourne Parkville Vic. Australia

4. Department of Cardiometabolic Health The University of Melbourne Parkville Vic. Australia

5. ACRF Rational Drug Discovery Centre St. Vincent's Institute of Medical Research Fitzroy Vic. Australia

6. School of Health and Biomedical Sciences RMIT University Melbourne Vic. Australia

7. Centre for Integrative Signalling Analysis CISA University of Freiburg Freiburg Germany

8. Centre for Inflammatory Disease Imperial College London London UK

Funder

Australian Cancer Research Foundation

Deutsche Forschungsgemeinschaft

National Health and Medical Research Council

National Heart Foundation of Australia

Publisher

EMBO

Subject

Molecular Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.15252/emmm.202216236

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5. The Proinflammatory CD14+CD16+DR++Monocytes Are a Major Source of TNF

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