Targeting YAP to overcome acquired resistance to ALK inhibitors in ALK ‐rearranged lung cancer

Author:

Yun Mi Ran12ORCID,Choi Hun Mi2,Lee You Won2,Joo Hyeong Seok1,Park Chae Won2,Choi Jae Woo34,Kim Dong Hwi1,Kang Han Na12,Pyo Kyoung‐Ho2,Shin Eun Joo2,Shim Hyo Sup5,Soo Ross A6,Yang James Chih‐Hsin78,Lee Sung Sook9,Chang Hyun10,Kim Min Hwan2,Hong Min Hee2,Kim Hye Ryun2,Cho Byoung Chul2ORCID

Affiliation:

1. JEUK Institute for Cancer Research JEUK Co., Ltd. Gumi‐City Korea

2. Division of Medical Oncology Yonsei Cancer Center Yonsei University College of Medicine Seoul Korea

3. Department of Pharmacology Yonsei University College of Medicine Seoul Korea

4. Severance Biomedical Science Institute Yonsei University College of Medicine Seoul Korea

5. Department of Pathology Yonsei University College of Medicine Seoul Korea

6. Department of Haematology‐Oncology National University Cancer Institute Singapore Singapore

7. Graduate Institute of Oncology National Taiwan University Taipei Taiwan

8. Department of Oncology National Taiwan University Hospital Taipei Taiwan

9. Department of Hematology‐Oncology Inje University Haeundae Paik Hospital Busan Korea

10. International St. Mary's Hospital College of Medicine Catholic Kwandong University Incheon Korea

Funder

National Research Foundation of Korea

Publisher

EMBO

Subject

Molecular Medicine

Reference56 articles.

1. ALK inhibitors in non‐small cell lung cancer: crizotinib and beyond;Awad MM;Clin Adv Hematol Oncol,2014

2. Photosensitizers and Photodynamic Therapy: Verteporfin

3. Photodynamic therapy with verteporfin (Visudyne): impact on ophthalmology and visual sciences;Bressler NM;Invest Ophthalmol Vis Sci,2000

4. The clinically used photosensitizer Verteporfin (VP) inhibits YAP-TEAD and human retinoblastoma cell growth in vitro without light activation

5. Functional genomics screen identifies YAP1 as a key determinant to enhance treatment sensitivity in lung cancer cells

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