An EZH2‐dependent transcriptional complex promotes aberrant epithelial remodelling after injury-Reference-Cited by-同舟云学术

An EZH2‐dependent transcriptional complex promotes aberrant epithelial remodelling after injury

Published:2021-07-05 Issue:8 Volume:22 Page:
ISSN:1469-221X
Container-title:EMBO reports
language:en
Short-container-title:EMBO Rep

Author:

Le Huy Q¹^ORCID,Hill Matthew A¹²,Kollak Ines¹,Keck Martina¹,Schroeder Victoria¹,Wirth Johannes¹,Skronska‐Wasek Wioletta¹,Schruf Eva¹,Strobel Benjamin³^ORCID,Stahl Heiko¹,Herrmann Franziska E¹,Campos Alexandre R⁴,Li Jun⁵,Quast Karsten⁶,Knebel Dagmar⁶,Viollet Coralie⁶,Thomas Matthew J¹²,Lamb David¹,Garnett James P¹⁷

Affiliation:

1. Lung Repair & Regeneration Department Boehringer Ingelheim Pharma GmbH & Co. KG Biberach Germany

2. University of Bath Bath UK

3. Drug Discovery Sciences Boehringer Ingelheim Pharma GmbH & Co. KG Biberach Germany

4. Sanford Burnham Prebys Medical Discovery Institute La Jolla CA USA

5. Immunology and Respiratory Disease Research Department Boehringer Ingelheim Pharmaceuticals, Inc Ridgefield CT USA

6. Global Computational Biology and Digital Sciences Boehringer Ingelheim Pharma GmbH & Co. KG Biberach Germany

7. Translational and Clinical Research Institute Newcastle University Newcastle UK

Funder

Boehringer Ingelheim

Publisher

EMBO

Subject

Genetics,Molecular Biology,Biochemistry

Link

https://onlinelibrary.wiley.com/doi/pdf/10.15252/embr.202152785

Reference59 articles.

1. Unravelling the transcriptional responses of TGF‐β: Smad3 and EZH2 constitute a regulatory switch that controls neuroretinal epithelial cell fate specification

2. Ezh1 Targets Bivalent Genes to Maintain Self-Renewing Stem Cells in Ezh2-Insufficient Myelodysplastic Syndrome

3. Automatization and improvement of μCT analysis for murine lung disease models using a deep learning approach

4. Transcriptomic profiling reveals disease-specific characteristics of epithelial cells in idiopathic pulmonary fibrosis

5. Genome-wide mapping of Polycomb target genes unravels their roles in cell fate transitions

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