SCAI promotes error‐free repair of DNA interstrand crosslinks via the Fanconi anemia pathway-Reference-Cited by-同舟云学术

SCAI promotes error‐free repair of DNA interstrand crosslinks via the Fanconi anemia pathway

Published:2022-02-14 Issue:4 Volume:23 Page:
ISSN:1469-221X
Container-title:EMBO reports
language:en
Short-container-title:EMBO Reports

Author:

Schubert Lisa¹,Hendriks Ivo A²^ORCID,Hertz Emil P T¹,Wu Wei³,Sellés‐Baiget Selene¹,Hoffmann Saskia¹,Viswalingam Keerthana Stine⁴^ORCID,Gallina Irene¹^ORCID,Pentakota Satyakrishna¹,Benedict Bente¹^ORCID,Johansen Joachim⁵,Apelt Katja⁶,Luijsterburg Martijn S⁶,Rasmussen Simon⁵^ORCID,Lisby Michael³⁴^ORCID,Liu Ying³,Nielsen Michael L²,Mailand Niels¹³^ORCID,Duxin Julien P¹^ORCID

Affiliation:

1. Protein Signaling Program Novo Nordisk Foundation Center for Protein Research University of Copenhagen Copenhagen Denmark

2. Proteomics Program Novo Nordisk Foundation Center for Protein Research University of Copenhagen Copenhagen Denmark

3. Center for Chromosome Stability Department of Cellular and Molecular Medicine University of Copenhagen Copenhagen Denmark

4. Department of Biology University of Copenhagen Copenhagen Denmark

5. Disease Systems Biology Novo Nordisk Foundation Center for Protein Research University of Copenhagen Copenhagen Denmark

6. Department of Human Genetics Leiden University Medical Center Leiden The Netherlands

Funder

Lundbeckfonden

H2020 Marie Skłodowska-Curie Actions

H2020 European Research Council

Sundhed og Sygdom, Det Frie Forskningsråd

Novo Nordisk Fonden

Publisher

EMBO

Subject

Genetics,Molecular Biology,Biochemistry

Link

https://onlinelibrary.wiley.com/doi/pdf/10.15252/embr.202153639

Reference66 articles.

1. The Protexin complex counters resection on stalled forks to promote homologous recombination and crosslink repair

2. DNA Replication Is Required To Elicit Cellular Responses to Psoralen-Induced DNA Interstrand Cross-Links

3. A complex of BRCA2 and PP2A-B56 is required for DNA repair by homologous recombination

4. Replication Fork Reversal during DNA Interstrand Crosslink Repair Requires CMG Unloading

5. Human HMGN1 and HMGN2 are not required for transcription-coupled DNA repair

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