MOAP‐1‐mediated dissociation of p62/SQSTM1 bodies releases Keap1 and suppresses Nrf2 signaling-Reference-Cited by-同舟云学术

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MOAP‐1‐mediated dissociation of p62/SQSTM1 bodies releases Keap1 and suppresses Nrf2 signaling

Published:2021-01-04 Issue:1 Volume:22 Page:
ISSN:1469-221X
Container-title:EMBO reports
language:en
Short-container-title:EMBO Rep

Author:

Tan Chong Teik¹^ORCID,Chang Hao‐Chun¹^ORCID,Zhou Qiling¹²,Yu Chundong²^ORCID,Fu Nai Yang³,Sabapathy Kanaga³⁴,Yu Victor C¹^ORCID

Affiliation:

1. Department of Pharmacy National University of Singapore Singapore Singapore

2. School of Life Sciences Xiamen University Xiamen China

3. Cancer & Stem Cell Biology Program Duke‐NUS Medical School Singapore Singapore

4. Division of Cellular & Molecular Research Humphrey Oei Institute of Cancer Research National Cancer Centre Singapore Singapore

Publisher

EMBO

Subject

Genetics,Molecular Biology,Biochemistry

Link

https://onlinelibrary.wiley.com/doi/pdf/10.15252/embr.202050854

Reference64 articles.

1. Considerations and Challenges in Studying Liquid-Liquid Phase Separation and Biomolecular Condensates

2. Lgr5+ pericentral hepatocytes are self-maintained in normal liver regeneration and susceptible to hepatocarcinogenesis

3. The Tumor Suppressor RASSF1A and MAP-1 Link Death Receptor Signaling to Bax Conformational Change and Cell Death

4. p62/SQSTM1 forms protein aggregates degraded by autophagy and has a protective effect on huntingtin-induced cell death

5. Oxidation of SQSTM1/p62 mediates the link between redox state and protein homeostasis

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