The UPR sensor IRE1α promotes dendritic cell responses to control Toxoplasma gondii infection-Reference-Cited by-同舟云学术

The UPR sensor IRE1α promotes dendritic cell responses to control Toxoplasma gondii infection

Published:2021-02-15 Issue:3 Volume:22 Page:
ISSN:1469-221X
Container-title:EMBO reports
language:en
Short-container-title:EMBO Rep

Author:

Poncet Anaïs F¹,Bosteels Victor²³,Hoffmann Eik¹^ORCID,Chehade Sylia¹,Rennen Sofie²³^ORCID,Huot Ludovic¹,Peucelle Véronique¹,Maréchal Sandra²³,Khalife Jamal¹^ORCID,Blanchard Nicolas⁴^ORCID,Janssens Sophie²³^ORCID,Marion Sabrina¹^ORCID

Affiliation:

1. Univ. Lille, CNRS, INSERM, CHU Lille Institut Pasteur de Lille U1019 ‐ UMR 9017 ‐ CIIL ‐ Center for Infection and Immunity of Lille Lille France

2. Laboratory for ER stress and Inflammation VIB Center for Inflammation Research Ghent Belgium

3. Department of Internal Medicine and Pediatrics Ghent University Ghent Belgium

4. Centre de Physiopathologie Toulouse Purpan (CPTP) INSERM, CNRS, UPS Université de Toulouse Toulouse France

Funder

Agence Nationale de la Recherche

H2020 European Research Council

Publisher

EMBO

Subject

Genetics,Molecular Biology,Biochemistry

Link

https://onlinelibrary.wiley.com/doi/pdf/10.15252/embr.201949617

Reference65 articles.

1. Combined Action of Nucleic Acid-Sensing Toll-like Receptors and TLR11/TLR12 Heterodimers Imparts Resistance to Toxoplasma gondii in Mice

2. Toxoplasma gondii Co-opts the Unfolded Protein Response To Enhance Migration and Dissemination of Infected Host Cells

3. Persistence ofToxoplasma gondiiin the central nervous system: a fine-tuned balance between the parasite, the brain and the immune system

4. Cleavage of BLOC1S1 mRNA by IRE1 Is Sequence Specific, Temporally Separate from XBP1 Splicing, and Dispensable for Cell Viability under Acute Endoplasmic Reticulum Stress

5. Sec22b Regulates Phagosomal Maturation and Antigen Crosspresentation by Dendritic Cells

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