ER stress in antigen‐presenting cells promotes NKT cell activation through endogenous neutral lipids-Reference-Cited by-同舟云学术

ER stress in antigen‐presenting cells promotes NKT cell activation through endogenous neutral lipids

Published:2020-05-03 Issue:6 Volume:21 Page:
ISSN:1469-221X
Container-title:EMBO reports
language:en
Short-container-title:EMBO Reports

Author:

Govindarajan Srinath¹²,Verheugen Eveline¹²,Venken Koen¹²,Gaublomme Djoere¹²,Maelegheer Margaux¹²,Cloots Eva²³⁴,Gysens Fien⁵⁶,De Geest Bruno G⁶,Cheng Tan‐Yun⁷,Moody D Branch⁷,Janssens Sophie²³,Drennan Michael¹²,Elewaut Dirk¹²^ORCID

Affiliation:

1. Unit for Molecular Immunology and Inflammation VIB‐Center for Inflammation Research Ghent Belgium

2. Department of Internal Medicine and Pediatrics Ghent University Ghent Belgium

3. Laboratory for ER Stress and Inflammation VIB‐Center for Inflammation Research Ghent Belgium

4. VIB‐Center for Medical Biotechnology Ghent Belgium

5. Department of Biomolecular Medicine Center for Medical Genetics Ghent University Ghent Belgium

6. Biopharmaceutical Technology Unit Department of Pharmaceutics Ghent University Ghent Belgium

7. Brigham and Women's Hospital Division of Rheumatology Immunity and Inflammation Harvard Medical School Boston MA USA

Funder

Fonds Wetenschappelijk Onderzoek

Stichting Tegen Kanker

National Institutes of Health

Publisher

EMBO

Subject

Genetics,Molecular Biology,Biochemistry

Link

https://onlinelibrary.wiley.com/doi/pdf/10.15252/embr.201948927

Reference73 articles.

1. The unfolded protein response: controlling cell fate decisions under ER stress and beyond

2. The Unfolded Protein Response: From Stress Pathway to Homeostatic Regulation

3. Signal integration in the endoplasmic reticulum unfolded protein response

4. XBP-1 is required for biogenesis of cellular secretory machinery of exocrine glands

5. IRE1 couples endoplasmic reticulum load to secretory capacity by processing the XBP-1 mRNA

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