Dipeptidyl peptidase 9 triggers <scp>BRCA2</scp> degradation and promotes <scp>DNA</scp> damage repair-Reference-Cited by-同舟云学术

Dipeptidyl peptidase 9 triggers BRCA2 degradation and promotes DNA damage repair

Published:2022-08 Issue:10 Volume:23 Page:
ISSN:1469-221X
Container-title:EMBO reports
language:en
Short-container-title:EMBO Reports

Author:

Bolgi Oguz¹²^ORCID,Silva‐Garcia Maria²,Ross Breyan³⁴,Pilla Esther²,Kari Vijayalakshmi⁵,Killisch Markus²,Spitzner Melanie⁵,Stark Nadine⁶,Lenz Christof⁷⁸^ORCID,Weiss Konstantin⁹,Donzelli Laura¹^ORCID,Gorrell Mark D¹⁰^ORCID,Grade Marian⁵,Riemer Jan⁹^ORCID,Urlaub Henning⁷⁸^ORCID,Dobbelstein Matthias⁶^ORCID,Huber Robert³¹¹¹²,Geiss‐Friedlander Ruth¹²^ORCID

Affiliation:

1. Institute of Molecular Medicine and Cell Research, Medical Faculty University of Freiburg Freiburg Germany

2. Department of Molecular Biology University Medical Center Göttingen Göttingen Germany

3. Max Planck Institut für Biochemie Martinsried Germany

4. Proteros Biostructures GmbH Martinsried Germany

5. Department of General, Visceral and Pediatric Surgery University Medical Center Göttingen Göttingen Germany

6. Institute of Molecular Oncology, Göttingen Center of Molecular Biosciences (GZMB) University Medical Center Göttingen Göttingen Germany

7. Bioanalytics, Institute of Clinical Chemistry University Medical Center Göttingen Germany

8. Bioanalytical Mass Spectrometry Group Max Planck Institute for Biophysical Chemistry Göttingen Germany

9. Institute of Biochemistry, Redox Biochemistry, and Cologne Excellence Cluster on Cellular Stress Responses in Aging‐Associated Diseases (CECAD) University of Cologne Cologne Germany

10. Centenary Institute The University of Sydney Faculty of Medicine and Health Sydney NSW Australia

11. Zentrum für Medizinische Biotechnologie Universität Duisburg‐Essen Essen Germany

12. Fakultät für Chemie Technische Universität München Garching Germany

Funder

Deutsche Forschungsgemeinschaft

Publisher

EMBO

Subject

Genetics,Molecular Biology,Biochemistry

Link

https://onlinelibrary.wiley.com/doi/pdf/10.15252/embr.202154136

Reference117 articles.

1. High levels of BRC4 induced by a Tet-On 3G system suppress DNA repair and impair cell proliferation in vertebrate cells

2. Dipeptidyl peptidase 9 has two forms, a broad tissue distribution, cytoplasmic localization and DPIV-like peptidase activity

3. Proteomics analyses reveal the evolutionary conservation and divergence of N-terminal acetyltransferases from yeast and humans

4. The Carboxyl Terminus of Brca2 Links the Disassembly of Rad51 Complexes to Mitotic Entry

5. In Vivo Half-Life of a Protein Is a Function of Its Amino-Terminal Residue

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