CDC7 kinase promotes MRE11 fork processing, modulating fork speed and chromosomal breakage-Reference-Cited by-同舟云学术

CDC7 kinase promotes MRE11 fork processing, modulating fork speed and chromosomal breakage

Published:2020-06-04 Issue:8 Volume:21 Page:
ISSN:1469-221X
Container-title:EMBO reports
language:en
Short-container-title:EMBO Rep

Author:

Rainey Michael D¹^ORCID,Quinlan Aisling¹^ORCID,Cazzaniga Chiara¹^ORCID,Mijic Sofija²,Martella Oliviano¹,Krietsch Jana²,Göder Anja¹^ORCID,Lopes Massimo²^ORCID,Santocanale Corrado¹^ORCID

Affiliation:

1. Centre for Chromosome Biology School of Natural Sciences National University of Ireland Galway Galway Ireland

2. Institute of Molecular Cancer Research University of Zurich Zurich Switzerland

Funder

Science Foundation Ireland

H2020 European Research Council

Krebsliga Schweiz

Publisher

EMBO

Subject

Genetics,Molecular Biology,Biochemistry

Link

https://onlinelibrary.wiley.com/doi/pdf/10.15252/embr.201948920

Reference75 articles.

1. On the mechanism of DNA replication in mammalian chromosomes

2. Autoradiography of HeLa cell DNA

3. Phosphorylation of MCM4 by Cdc7 Kinase Facilitates Its Interaction with Cdc45 on the Chromatin

4. Identification of Mcm2 Phosphorylation Sites by S-phase-regulating Kinases

5. Cdc7-Dbf4 Phosphorylates MCM Proteins via a Docking Site-Mediated Mechanism to Promote S Phase Progression

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2. DBF4, not DRF1, is the crucial regulator of CDC7 kinase at replication forks;Journal of Cell Biology;2024-06-12

3. Dbf4-dependent kinase promotes cell cycle controlled resection of DNA double-strand breaks and repair by homologous recombination;Nature Communications;2024-04-03

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