Nox4 regulates InsP 3 receptor‐dependent Ca 2+ release into mitochondria to promote cell survival-Reference-Cited by-同舟云学术

Nox4 regulates InsP 3 receptor‐dependent Ca 2+ release into mitochondria to promote cell survival

Published:2020-08-10 Issue:19 Volume:39 Page:
ISSN:0261-4189
Container-title:The EMBO Journal
language:en
Short-container-title:EMBO J

Author:

Beretta Matteo¹,Santos Celio XC¹,Molenaar Chris¹,Hafstad Anne D¹²,Miller Chris CJ³^ORCID,Revazian Aram⁴,Betteridge Kai¹,Schröder Katrin⁵^ORCID,Streckfuß‐Bömeke Katrin⁶^ORCID,Doroshow James H⁷,Fleck Roland A⁸,Su Tsung‐Ping⁹,Belousov Vsevolod V⁴¹⁰¹¹,Parsons Maddy¹²^ORCID,Shah Ajay M¹^ORCID

Affiliation:

1. School of Cardiovascular Medicine & Sciences King's College London British Heart Foundation Centre London UK

2. Cardiovascular Research Group Department of Medical Biology UIT‐The Arctic University of Norway Tromsø Norway

3. Department of Basic and Clinical Neuroscience Institute of Psychiatry, Psychology and Neuroscience King's College London London UK

4. Institute for Cardiovascular Physiology Georg August University Göttingen Göttingen Germany

5. Institute for Cardiovascular Physiology Goethe‐University Frankfurt Frankfurt am Main Germany

6. Department of Cardiology and Pneumology Universitätsmedizin Göttingen Göttingen Germany

7. Division of Cancer Treatment and Diagnosis National Cancer Institute NIH Bethesda MD USA

8. Centre for Ultrastructural Imaging King's College London London UK

9. Cellular Pathobiology Section National Institute on Drug Abuse, NIH Baltimore MD USA

10. Shemyakin‐Ovchinnikov Institute of Bioorganic Chemistry Moscow Russia

11. Pirogov Russian National Research Medical University Moscow Russia

12. King's College London British Heart Foundation Centre Randall Centre of Cell and Molecular Biophysics London UK

Funder

Fondation Leducq

Arthritis Research UK

California Department of Fish and Game

Publisher

EMBO

Subject

General Immunology and Microbiology,General Biochemistry, Genetics and Molecular Biology,Molecular Biology,General Neuroscience

Link

https://onlinelibrary.wiley.com/doi/pdf/10.15252/embj.2019103530

Reference53 articles.

1. Direct Interaction of the Novel Nox Proteins with p22phox Is Required for the Formation of a Functionally Active NADPH Oxidase

2. Nox4 and Nox2 NADPH Oxidases Mediate Distinct Cellular Redox Signaling Responses to Agonist Stimulation

3. Loss of cyclophilin D reveals a critical role for mitochondrial permeability transition in cell death

4. Nox4 NADPH oxidase-derived reactive oxygen species, via endogenous carbon monoxide, promote survival of brain endothelial cells during TNF-α-induced apoptosis

5. Genetically encoded fluorescent indicator for intracellular hydrogen peroxide

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