Differential retrotranslocation of mitochondrial Bax and Bak-Reference-Cited by-同舟云学术

Differential retrotranslocation of mitochondrial Bax and Bak

Published:2014-11-05 Issue:1 Volume:34 Page:67-80
ISSN:0261-4189
Container-title:The EMBO Journal
language:en
Short-container-title:EMBO J

Author:

Todt Franziska¹²³,Cakir Zeynep¹³,Reichenbach Frank¹²³,Emschermann Frederic⁴⁵,Lauterwasser Joachim¹³,Kaiser Andrea¹,Ichim Gabriel⁶,Tait Stephen WG⁶,Frank Stephan⁷,Langer Harald F⁴⁵,Edlich Frank¹²⁸

Affiliation:

1. Institute for Biochemistry and Molecular Biology University of Freiburg Freiburg Germany

2. Spemann Graduate School of Biology and Medicine SGBM Freiburg Germany

3. Faculty of Biology University of Freiburg Freiburg Germany

4. Section for Cardioimmunology University of Tübingen Tübingen Germany

5. Department of Cardiovascular Medicine University Hospital University of Tübingen Tübingen Germany

6. Cancer Research UK Beatson Institute University of Glasgow Glasgow UK

7. Division of Neuropathology Institute of Pathology Basel University Hospitals Basel Switzerland

8. BIOSS Centre for Biological Signaling Studies University of Freiburg Freiburg Germany

Publisher

EMBO

Subject

General Immunology and Microbiology,General Biochemistry, Genetics and Molecular Biology,Molecular Biology,General Neuroscience

Link

https://onlinelibrary.wiley.com/doi/pdf/10.15252/embj.201488806

Reference45 articles.

1. Bax oligomerization is required for channel-forming activity in liposomes and to trigger cytochrome c release from mitochondria

2. A Rapid, Reversible, and Tunable Method to Regulate Protein Function in Living Cells Using Synthetic Small Molecules

3. Caspase Cascades in Chemically-Induced Apoptosis

4. Transient binding of an activator BH3 domain to the Bak BH3-binding groove initiates Bak oligomerization

5. Bid-induced Conformational Change of Bax Is Responsible for Mitochondrial Cytochrome c Release during Apoptosis

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