NSUN 3 and ABH 1 modify the wobble position of mt‐t RNA Met to expand codon recognition in mitochondrial translation-Reference-Cited by-同舟云学术

NSUN 3 and ABH 1 modify the wobble position of mt‐t RNA Met to expand codon recognition in mitochondrial translation

Published:2016-08-06 Issue:19 Volume:35 Page:2104-2119
ISSN:0261-4189
Container-title:The EMBO Journal
language:en
Short-container-title:EMBO J

Author:

Haag Sara¹,Sloan Katherine E¹,Ranjan Namit²,Warda Ahmed S¹,Kretschmer Jens¹,Blessing Charlotte¹,Hübner Benedikt¹,Seikowski Jan³⁴,Dennerlein Sven⁵,Rehling Peter⁴⁵⁶,Rodnina Marina V²,Höbartner Claudia³^ORCID,Bohnsack Markus T¹⁶

Affiliation:

1. Institute for Molecular Biology University Medical Center Göttingen Georg‐August‐University Göttingen Germany

2. Department of Physical Biochemistry Max Planck Institute for Biophysical Chemistry Göttingen Germany

3. Institute for Organic and Biomolecular Chemistry Georg‐August‐University Göttingen Germany

4. Max Planck Institute for Biophysical Chemistry Göttingen Germany

5. Institute for Cellular Biochemistry University Medical Center Göttingen Georg‐August‐University Göttingen Germany

6. Göttingen Centre for Molecular Biosciences Georg‐August‐University Göttingen Germany

Funder

Deutsche Forschungsgemeinschaft

Alexander von Humboldt-Stiftung

European Research Council

Georg-August-Universität Göttingen

Max-Planck-Gesellschaft

Publisher

EMBO

Subject

General Immunology and Microbiology,General Biochemistry, Genetics and Molecular Biology,Molecular Biology,General Neuroscience

Link

https://onlinelibrary.wiley.com/doi/pdf/10.15252/embj.201694885

Reference84 articles.

1. Human and bacterial oxidative demethylases repair alkylation damage in both RNA and DNA

2. tRNA’s Wobble Decoding of the Genome: 40 Years of Modification

3. FTO levels affect RNA modification and the transcriptome

4. The Human Mitochondrial tRNAMet: Structure/Function Relationship of a Unique Modification in the Decoding of Unconventional Codons

5. Aberrant methylation of t RNA s links cellular stress to neuro‐developmental disorders

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