Emancipation from transcriptional latency: unphosphorylated STAT5 as guardian of hematopoietic differentiation-Reference-Cited by-同舟云学术

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Emancipation from transcriptional latency: unphosphorylated STAT5 as guardian of hematopoietic differentiation

Published:2016-02-18 Issue:6 Volume:35 Page:555-557
ISSN:0261-4189
Container-title:The EMBO Journal
language:en
Short-container-title:EMBO J

Author:

Decker Thomas¹

Affiliation:

1. Max F. Perutz Laboratories University of Vienna Vienna Austria

Funder

Austrian Science Foundation (FWF)

Publisher

EMBO

Subject

General Immunology and Microbiology,General Biochemistry, Genetics and Molecular Biology,Molecular Biology,General Neuroscience

Link

https://onlinelibrary.wiley.com/doi/pdf/10.15252/embj.201693974

Reference12 articles.

1. Mechanisms of cohesin-mediated gene regulation and lessons learned from cohesinopathies

2. The Functions of Signal Transducers and Activators of Transcriptions 1 and 3 as Cytokine-Inducible Proteins

3. Nongenomic Functions of STAT3

4. Unphosphorylated STAT5A stabilizes heterochromatin and suppresses tumor growth

5. STAT signaling in Dictyostelium development

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1. The structural influence of the oncogenic driver mutation N642H in the STAT5B SH2 domain;2024-09-10

2. Small molecule STAT3/5 inhibitors exhibit therapeutic potential in acute myeloid leukemia and extra-nodal natural killer/T cell lymphoma;2023-10-18

3. Small molecule STAT3/5 inhibitors exhibit therapeutic potential in acute myeloid leukemia and extra-nodal natural killer/T cell lymphoma;2023-10-16

4. STAT proteins: a kaleidoscope of canonical and non-canonical functions in immunity and cancer;Journal of Hematology & Oncology;2021-11-22

5. Translational and clinical advances in JAK-STAT biology: The present and future of jakinibs;Journal of Leukocyte Biology;2018-07-12

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