Uncoupling endosomal CLC chloride/proton exchange causes severe neurodegeneration-Reference-Cited by-同舟云学术

Uncoupling endosomal CLC chloride/proton exchange causes severe neurodegeneration

Published:2020-03-02 Issue:9 Volume:39 Page:
ISSN:0261-4189
Container-title:The EMBO Journal
language:en
Short-container-title:EMBO J

Author:

Weinert Stefanie¹²,Gimber Niclas¹²^ORCID,Deuschel Dorothea¹²,Stuhlmann Till¹²,Puchkov Dmytro¹,Farsi Zohreh²,Ludwig Carmen F¹²,Novarino Gaia¹²,López‐Cayuqueo Karen I¹²,Planells‐Cases Rosa¹²,Jentsch Thomas J¹²³^ORCID

Affiliation:

1. Leibniz‐Forschungsinstitut für Molekulare Pharmakologie (FMP) Berlin Germany

2. Max‐Delbrück‐Centrum für Molekulare Medizin (MDC) Berlin Germany

3. NeuroCure Cluster of Excellence Charité Universitätsmedizin Berlin Berlin Germany

Funder

Deutsche Forschungsgemeinschaft

H2020 European Research Council

Louis-Jeantet Foundation

Publisher

EMBO

Subject

General Immunology and Microbiology,General Biochemistry, Genetics and Molecular Biology,Molecular Biology,General Neuroscience

Link

https://onlinelibrary.wiley.com/doi/pdf/10.15252/embj.2019103358

Reference96 articles.

1. Secondary active transport mediated by a prokaryotic homologue of ClC Cl- channels

2. Proteomic screening of glutamatergic mouse brain synaptosomes isolated by fluorescence activated sorting

3. A novel CLCN5 pathogenic mutation supports Dent disease with normal endosomal acidification

4. Molecular Profiling of Synaptic Vesicle Docking Sites Reveals Novel Proteins but Few Differences between Glutamatergic and GABAergic Synapses

5. The yeast CLC protein counteracts vesicular acidification during iron starvation

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