TGIF 1 functions as a tumor suppressor in pancreatic ductal adenocarcinoma-Reference-Cited by-同舟云学术

TGIF 1 functions as a tumor suppressor in pancreatic ductal adenocarcinoma

Published:2019-05-31 Issue:13 Volume:38 Page:
ISSN:0261-4189
Container-title:The EMBO Journal
language:en
Short-container-title:EMBO J

Author:

Parajuli Parash¹,Singh Purba²,Wang Zhe²,Li Lianna²,Eragamreddi Sailaja²,Ozkan Seval²^ORCID,Ferrigno Olivier³,Prunier Celine³,Razzaque Mohammed S⁴,Xu Keli²,Atfi Azeddine¹³^ORCID

Affiliation:

1. Cellular and Molecular Pathogenesis Division Department of Pathology and Massey Cancer Center Virginia Commonwealth University Richmond VA USA

2. Cancer Institute University of Mississippi Medical Center Jackson MS USA

3. Centre de Recherche Saint‐Antoine, CRSA Inserm Sorbonne Universités Paris France

4. Department of Pathology Lake Erie College of Osteopathic Medicine Erie PA USA

Funder

National Institutes of Health

U.S. Department of Defense

Association pour la Recherche sur le Cancer

Publisher

EMBO

Subject

General Immunology and Microbiology,General Biochemistry, Genetics and Molecular Biology,Molecular Biology,General Neuroscience

Link

https://onlinelibrary.wiley.com/doi/pdf/10.15252/embj.2018101067

Reference61 articles.

1. Activated Kras and Ink4a/Arf deficiency cooperate to produce metastatic pancreatic ductal adenocarcinoma

2. Most human carcinomas of the exocrine pancreas contain mutant c-K-ras genes

3. Smads as transcriptional co-modulators

4. Signal Transduction by the TGF-β Superfamily

5. Both p16Ink4a and the p19Arf-p53 pathway constrain progression of pancreatic adenocarcinoma in the mouse

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