A new sub‐pathway of long‐patch base excision repair involving 5′ gap formation-Reference-Cited by-同舟云学术

A new sub‐pathway of long‐patch base excision repair involving 5′ gap formation

Published:2017-04-03 Issue:11 Volume:36 Page:1605-1622
ISSN:0261-4189
Container-title:The EMBO Journal
language:en
Short-container-title:EMBO J

Author:

Woodrick Jordan¹,Gupta Suhani¹,Camacho Sharon¹,Parvathaneni Swetha²,Choudhury Sujata¹,Cheema Amrita¹,Bai Yi¹,Khatkar Pooja¹,Erkizan Hayriye Verda¹,Sami Furqan²,Su Yan³,Schärer Orlando D³,Sharma Sudha²^ORCID,Roy Rabindra¹^ORCID

Affiliation:

1. Department of Oncology Lombardi Comprehensive Cancer Center Georgetown University Washington DC USA

2. Department of Biochemistry and Molecular Biology College of Medicine Howard University Washington DC USA

3. Department of Pharmacological Sciences & Department of Chemistry Stony Brook University Stony Brook NY USA

Funder

National Institutes of Health

Publisher

EMBO

Subject

General Immunology and Microbiology,General Biochemistry, Genetics and Molecular Biology,Molecular Biology,General Neuroscience

Link

https://onlinelibrary.wiley.com/doi/pdf/10.15252/embj.201694920

Reference73 articles.

1. ATM Regulates 3-Methylpurine-DNA Glycosylase and Promotes Therapeutic Resistance to Alkylating Agents

2. ERCC1-XPF Endonuclease Facilitates DNA Double-Strand Break Repair

3. Extracts of proliferating and non-proliferating human cells display different base excision pathways and repair fidelity

4. Poly(ADP‐ribose) polymerase in base excision repair: always engaged, but not essential for DNA damage processing;Allinson SL;Acta Biochim Pol,2003

5. Oxidative DNA damage induced by potassium bromate under cell-free conditions and in mammalian cells

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