USP 18 lack in microglia causes destructive interferonopathy of the mouse brain-Reference-Cited by-同舟云学术

USP 18 lack in microglia causes destructive interferonopathy of the mouse brain

Published:2015-04-20 Issue:12 Volume:34 Page:1612-1629
ISSN:0261-4189
Container-title:The EMBO Journal
language:en
Short-container-title:EMBO J

Author:

Goldmann Tobias¹,Zeller Nicolas¹,Raasch Jenni¹,Kierdorf Katrin¹,Frenzel Kathrin¹,Ketscher Lars¹,Basters Anja¹,Staszewski Ori¹,Brendecke Stefanie M¹,Spiess Alena¹,Tay Tuan Leng¹,Kreutz Clemens²,Timmer Jens²³,Mancini Grazia MS⁴,Blank Thomas¹,Fritz Günter¹,Biber Knut⁵⁶,Lang Roland⁷,Malo Danielle⁸,Merkler Doron⁹,Heikenwälder Mathias¹⁰,Knobeloch Klaus‐Peter¹,Prinz Marco¹³

Affiliation:

1. Institute of Neuropathology University of Freiburg Freiburg Germany

2. Institute of Physics & Center for Systems Biology (ZBSA) University of Freiburg Freiburg Germany

3. BIOSS Centre for Biological Signalling Studies University of Freiburg Freiburg Germany

4. Department of Clinical Genetics Erasmus University Medical Center Rotterdam The Netherlands

5. Department of Psychiatry University of Freiburg Freiburg Germany

6. Department of Neuroscience University Medical Center Groningen Groningen The Netherlands

7. Institute of Clinical Microbiology, Immunology and Hygiene University Hospital Erlangen Erlangen Germany

8. Department of Human Genetics McGill University Montreal QC Canada

9. Department of Pathology and Immunology University of Geneva Geneva Switzerland

10. Institute of Virology Technische Universität München/Helmholtz‐Zentrum Munich München Germany

Publisher

EMBO

Subject

General Immunology and Microbiology,General Biochemistry, Genetics and Molecular Biology,Molecular Biology,General Neuroscience

Link

https://onlinelibrary.wiley.com/doi/pdf/10.15252/embj.201490791

Reference56 articles.

1. Genome-Wide DNA Methylation Patterns in Naive CD4+ T Cells From Patients With Primary Sjögren's Syndrome

2. Molecular characterization of ubiquitin-specific protease 18 reveals substrate specificity for interferon-stimulated gene 15

3. Loss of the cylindromatosis tumour suppressor inhibits apoptosis by activating NF-κB

4. Type I interferonopathies: Mendelian type I interferon up-regulation

5. The deubiquitinase activity of A 20 is dispensable for NF ‐κ B signaling

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