The FTD ‐like syndrome causing TREM 2 T66M mutation impairs microglia function, brain perfusion, and glucose metabolism

Author:

Kleinberger Gernot12ORCID,Brendel Matthias3,Mracsko Eva4,Wefers Benedikt56,Groeneweg Linda4,Xiang Xianyuan1,Focke Carola3,Deußing Maximilian3,Suárez‐Calvet Marc15,Mazaheri Fargol5ORCID,Parhizkar Samira1,Pettkus Nadine1,Wurst Wolfgang2567,Feederle Regina258,Bartenstein Peter23,Mueggler Thomas4,Arzberger Thomas5910,Knuesel Irene4,Rominger Axel23,Haass Christian125ORCID

Affiliation:

1. Biomedical Center (BMC), Biochemistry Ludwig‐Maximilians‐Universität München Munich Germany

2. Munich Cluster for Systems Neurology (SyNergy) Munich Germany

3. Department of Nuclear Medicine Ludwig‐Maximilians‐Universität München Munich Germany

4. NORD Discovery & Translational Area, Roche Pharmaceutical Research and Early Development Roche Innovation Center Basel Basel Switzerland

5. German Center for Neurodegenerative Diseases (DZNE) Munich Germany

6. Institute of Developmental Genetics Helmholtz Zentrum München German Research Center for Environmental Health Neuherberg Germany

7. Technische Universität München Freising‐Weihenstephan Germany

8. Helmholtz Center Munich, German Research Center for Environmental Health Institute for Diabetes and Obesity, Core Facility Monoclonal Antibody Development Munich Germany

9. Center for Neuropathology and Prion Research Ludwig‐Maximilians‐Universität München Munich Germany

10. Department of Psychiatry and Psychotherapy Ludwig‐Maximilians‐Universität München Munich Germany

Funder

Deutsche Forschungsgemeinschaft

H2020 European Research Council

Cure Alzheimer's Fund

Seventh Framework Programme

Publisher

EMBO

Subject

General Immunology and Microbiology,General Biochemistry, Genetics and Molecular Biology,Molecular Biology,General Neuroscience

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